Tuesday, February 19, 2013

Symptoms of Lou Gehrig Disease and Lou Gehrig Disease Treatment

Amyotrophic lateral sclerosis (A.L.S.) is a neurodegenerative motor neurons diseaseof the nerve cells in the brain and spinal cord that control voluntary muscle movement. ALS is also known as Lou Gehrig's disease. In ALS, nerve cells (neurons) waste away or die, and can no longer send messages to muscles. These early symptoms of Lou Gehrig's disease eventually lead to muscle weakening, twitching, and an inability to move the arms, legs, and body. The condition slowly gets worse. When the muscles in the chest area stop working, it becomes hard or impossible to breathe on one's own. While there is no known cure, there are a number of natural supplements one can take to slow the progression of Lou Gehrig's disease.

As far as possible causes of ALS, there have been studies linking the aluminum content of water supplies to Lou Gehrig's disease. Aluminum can interfere with phosphate absorption leading to hypophosphatemia. In addition, apoptosis is a distinct form of cell death controlled by an internally encoded suicide program.

Apoptosis also accounts for much of the pathology seen in Lou Gehrig's disease, which is marked by the loss of brain neurons. In specific, a part of the nerve axon is damaged, leading to proximal axonopathy. Proximal axonopathies are often caused by volatile organic compounds, which decrease the excitability of the neuron by stabilizing membranes and decreasing ion flux. Elevations in apoptosis or cell death seem to be related to a lack of production of the nerve growth factor and to free radical damage. Manipulation of this process of cell killing as well as supporting healthy immune system function helps in treating A.L.S... Long-term delivery of nerve growth factors will protect against programmed cell death in individuals suffering from Lou Gehrig's disease.

Individuals with a familial dominant form of amyotrophic lateral sclerosis possess a defective gene that decreases cytoplasmic superoxide dismutase (SOD) by 40%. The cytoplasmic form of SOD requires copper and zinc. Evidence for the role of SOD in preventing free radical damage comes from a variety of sources. Unlike mammalian enzymes, a coated, non-soy, legume-derived SOD resists the action of pancreatic enzymes and a double-blind, placebo-controlled study suggested that oral ingestion of this supplement can increase the activity of SOD within 4 hours.

There is a strong correlation to heavy metal toxicity and neurological diseases. If you suspect heavy metal toxicity, a heavy metal toxicity detoxification program is in order.

Enough cannot be said about the importance of essential fatty acids in supporting a healthy nervous system. The fatty acids that are necessary for health and that cannot be made by the body are referred to as essential fatty acids (EFAs). EFAs aid in the transmission of nerve impulses and are needed for the normal development and functioning of the brain. A number of sources of essential fatty acids exist; among them are fish oils, flaxseeds and flaxseed oil, grape seed oil, and primrose oil.

In summary, if you or someone you love, has been given a diagnosis of A.L.S. or Lou Gehrig's disease, you may benefit from supplementation with S.O.D., zinc, copper and essential fatty acids, as well as undergoing a heavy metal detoxification program.

Dr. Stacy Foster is a naturopathic physician who graduated from an accredited university. Dr. Foster has been in clinical practice for over eleven years addressing all types of disease and ailments. If you would like to receive an absolutely FREE naturopathic medical consultation, please click on the link below.

Article Source: http://EzineArticles.com/?expert=Stacy_Foster

1 comment:

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